We analyze an evolutionary model with a finite number of players and with noise or mutations.The expansion and contraction of strategies is linked-as usual-to their current relative success, but mutations-which perturb the system away from its deterministic evolution-are present as well.Mutations can occur in every period, so the focus is on the implications of ongoing mutations, not a one-shot mutation.The effect of these mutations is to drastically reduce the set of equilibria to what we term "long-run equilibria."For 2 x 2 symmetric games with two symmetric strict Nash equilibria the equilibrium selected satisfies (for large populations) Harsanyi and Selten's (1988) criterion of risk-dominance.In particular, if both strategies have equal security levels, the Pareto dominant Nash equilibrium is selected, even though there is another strict Nash equilibrium.
This paper provides a simulated moments estimator (SME) of the parameters of dynamic models in which the state vector follows a time-homogeneous Markov process. Conditions are provided for both weak and strong consistency as well as asymptotic normality. Various tradeoff's among the regularity conditions underlying the large sample properties of the SME are discussed in the context of an asset pricing model.
Different extensive form games with the same reduced normal form can have different information sets and subgames. This generates a tension between a belief in the strategic relevance of information sets and subgames and a belief in the sufficiency of the reduced normal form. We identify a property of extensive form information sets and subgames which we term strategic independence. Strategic independence is captured by the reduced normal form, and can be used to define normal form information sets and subgames. We prove a close relationship between these normal form structures and their extensive form namesakes. Using these structures, we are able to motivate and implement solution concepts corresponding to subgame perfection, sequential equilibrium, and forward induction entirely in the reduced normal form, and show close relations between their implications in the normal and extensive form.